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In rats, anabolic steroids also act in the peripheral metabolism of thyroid hormones and seem to exert an important proliferative effect on thyroid cells. Effect upon T3 receptor structure and function Testosterone can regulate the activity of the T3 receptor which is involved in the conversion of T3 into T3-binding protein and thus regulate the level of plasma testosterone. The levels of T3 are also influenced by the thyroid hormone receptor itself in the hypothalamus – both within the hypothalamus and in the pituitary, where it is known that T3 is mainly produced and secreted by the thyroid. The effect of anabolic steroids upon the concentration of T3 in the bloodstream may be mediated by the influence of the T3 receptor on the concentration of T3-binding protein in the tissues. Although the role of anabolic steroids in anabolic muscle hypertrophy and catabolism still needs to be investigated, the hypothesis of that T3 receptors in muscle growth in particular can be considered in the framework of the recent results obtained from the study in rats. Further studies in this field are thus necessary, to establish this hypothesis and the mechanisms by which anabolic steroids promote their anabolic effects. The steroid hormones exert multiple actions on the cellular metabolism in terms of protein synthesis and cellular death of cell, both of which are involved in the regulation of protein metabolism. Anabolic steroids enhance the activity of the transcription factor and activator of transcription (STAT3), TNF-κB, resulting in the induction of genes involved in cell proliferation, proliferation and survival. Stat3 is also a regulator of cellular metabolism through binding to anabolic hormones in the plasma membrane. This regulation by Stat3 is critical to the process of protein synthesis. Anabolic steroids, in conjunction with anabolic hormones in the gut, are able to stimulate lipolysis in the muscles. There are other actions of anabolic steroids, although the exact mechanisms underlying this are still poorly understood. The inhibition of the enzymes known as acetyl-CoA dehydrogenase, which are highly involved in the conversion of fatty acids into triglycerides and thus lipolysis (lipotoxicity of both anabolic steroids and anabolic androgenic hormones), can be induced by anabolic steroids. Lipolysis of fatty acids in the muscle is important to provide the energy for muscle growth in both humans and rats, and therefore to increase the concentration of the anabolic androgenic hormones. A number of enzymes responsible for the conversion of fatty acids into triglycerides, such as palmitoylCoA-carnitine palmitoylCoA carboxylCoA carboxyl Co Similar articles: